Murraya koenigii = Curry tree

Anti-tumour promoting activity and antioxidant properties
of girinimbine isolated from the stem bark of Murraya koenigii S.

http://www.mdpi.com/1420-3049/17/4/4651




Coleus tuberosus = Plectranthus rotundifolius = Chinese potato = ubi kembili

http://www.globinmed.com/index.php?option=...gel&catid=380:p

Chemopreventive properties of phytosterols and maslinic acid extracted from Coleus tuberosus in inhibiting the expression of EBV early-antigen in Raji cells.

http://www.ncbi.nlm.nih.gov/m/pubmed/20491082




Anti-tumour promoter activity in Malaysian ginger rhizobia
used in traditional medicine.

http://www.nature.com/bjc/journal/v80/n1/abs/6690329a.html




Screening for the In Vitro Anti-tumor-promoting Activities of Edible Plants from Malaysia.


'' Further dilution experiments indicated an extract from the leaves of
Piper betle L. (Piperaceae) to be one of the most promising species. ''

Piper betle = Betel leaf

https://www.jstage.jst.go.jp/article/bbb/64...64_1_9/_article




This post has been edited by Tham: Jun 11 2014, 07:45 AM

The Chinese are at high risk of nasopharygeal cancer due to three main factors :

Genetics, mainly chromosome 6p21.3
EBV infection
Diet - salt fish


The virus resides in the nasal epithelium.

The standard antibody tests which you find in the labs here are :

IgA-VCA
IgA-EA

IgG-VCA
IgG-EA


For improved sensitivity and specificity, ask the lab if they test for the viral DNA.


Comparison of plasma Epstein-Barr virus (EBV) DNA levels and serum
EBV immunoglobulin A/virus capsid antigen antibody titers in patients
with nasopharyngeal carcinoma.

http://www.ncbi.nlm.nih.gov/pubmed/15022282



Improved accuracy of detection of nasopharyngeal carcinoma by combined
application of circulating Epstein-Barr virus DNA and anti-Epstein-Barr viral
capsid antigen IgA antibody.

http://www.clinchem.org/content/50/2/339.long




Etiological factors of nasopharyngeal carcinoma.

'' In the high risk population, genetic susceptibility to NPC has been mapped
to the HLA loci and adjacent genes in MHC region on chromosome 6p21. ''

http://www.ncbi.nlm.nih.gov/m/pubmed/24630258



Association between human leukocyte antigen and nasopharyngeal-carcinoma.

http://www.ncbi.nlm.nih.gov/m/pubmed/23262096



Genome-wide association study reveals multiple nasopharyngeal
carcinoma-associated loci within the HLA region at chromosome 6p21.3.

http://www.cell.com/ajhg/pdf/S0002-9297(09)00298-5.pdf






Make sure you don't eat salt fish or other salt-preserved foods.

NPC is endemic amongst the boat people of Hongkong for this very reason.



Early Detection of Nasopharyngeal Carcinoma.

'' NPC presents as a complex disease caused by an interaction between chronic
infection with oncogenic gamma herpesvirus Epstein-Barr virus (EBV) and
environmental and genetic factors, involving a multistep carcinogenic process.''


'' Nonviral exposure associated with the risk of NPC involves the consumption of
salt-preserved fish, a traditional staple food in several NPC-endemic areas. In studies
of Chinese populations, the relative risk of NPC associated with weekly consumption,
compared with no or rare consumption, generally ranged from 1.4 to 3.2 per 100,000
whereas that for daily consumption ranged from 1.8 to 7.5 .

Salt-preserved foods are a dietary staple in all NPC-endemic populations.

Thus, this dietary staple pattern may explain part of the international distribution
of NPC incidence. The carcinogenic potential of salt-preserved fish is supported by
experiments in rats, which develop malignant nasal and nasopharyngeal tumors
after salted fish consumption. The process of salt preservation is inefficient,
allowing fish and other foods to become partially putrefied. As a result, these
foods accumulate significant levels of nitrosamines, which are known carcinogens
in animals. Salt-preserved fish also contain bacterial mutagens, direct genotoxins,
and EBV-reacting substances, any or all of which could also contribute to the
observed association. However, there have been no prospective studies of NPC
risk associations with salt-preserved fish consumption, or virtually any other
environmental exposure, in endemic areas. ''


http://www.hindawi.com/journals/ijoto/2011/638058

I've already given you the studies above on the antiviral properties of
certain foods and plants but you don't seem to have noticed.

These others have antiviral properties. The studies are on Medline and elsewhere
if you know how to find them.


Garlic
EGCG
Monolaurin
Olive leaf extract
Lemon balm, or Melissa officinalis
Oregano oil
Elderberry


The above can be ordered as supplements from Iherb.



In vitro virucidal effects of Allium sativum (garlic) extract and compounds.

http://www.ncbi.nlm.nih.gov/m/pubmed/1470664



CMV is another member of the herpes family, and is a DNA virus like EBV, being
implicated in numerous diseases, including multiple sclerosis and some cancers.


Experimental study on the action of allitridin against human cytomegalovirus in vitro:
Inhibitory effects on immediate-early genes.

http://www.ncbi.nlm.nih.gov/m/pubmed/16844239/



Activities of Z-ajoene against tumour and viral spreading in vitro.

http://www.ncbi.nlm.nih.gov/m/pubmed/17521297/




Olive leaf's oleuropein and hydroxytyrosol compounds have activity against HIV.



Anti-HIV activity of olive leaf extract (OLE) and modulation of
host cell gene expression by HIV-1 infection and OLE treatment.

http://www.ncbi.nlm.nih.gov/m/pubmed/12878215/?



http://users.rcn.com/wussery/olive1.html

http://www.about-olive-leaf-extract.com/na...-antiviral.html

http://www.hepatitiscfree.com/olive_leaf_book.htm


http://www.researchgate.net/profile/Amparo...lication_detail



STUDIES ON THE EFFECT OF OLIVE LEAF EXTRACT
ON A MODEL OF DNA OR RNA VIRUSES.

http://www.researchgate.net/profile/Mohame...lication_detail


Inhibition of Epstein-Barr virus lytic cycle by (-)-epigallocatechin gallate.

http://www.ncbi.nlm.nih.gov/m/pubmed/12589821




(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous
lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells.

http://carcin.oxfordjournals.org/content/34/3/627.long


EGCG has activity against HIV as well, blocking the virus from binding to the gp120
receptor on your CD4 T-helper cells.


EGCG debilitates the persistence of EBV latency by reducing
the DNA binding potency of nuclear antigen 1.

http://www.ncbi.nlm.nih.gov/m/pubmed/22226960/




A new triple combination therapy.


http://www.ncbi.nlm.nih.gov/m/pubmed/11366542